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15.3 DNA Repair
The rate of mutation is measured in the other sample.
A t-test of these was observed.
You can reject the null hypothesis that the control by 2 million is the number of original cells.
You can accept the hypothesis that the suspected mutagen is caus -6.
This is a higher rate.
You are able to accept the hypothesis based on the statistical outcome.
In the presence of the mutagen, the rate of change is 20 times higher than the rate of change without it.
This research shows that urine from smokers has higher levels of mutagens.
The consequences and causes of mutations were considered in the previous sections.
We have seen that random events can have negative consequences.
A researcher can repair changes that occur in the structure of studied the effects of a suspected mutagen, DNA.
After placing 2 million cells on each plate, there is a proofreading function that helps to prevent data from being obtained.
Control with mutagen X and repair them.
People with xeroderma pigmentosum are susceptible to the harmful effects of sunlight because they are missing a 4 2 55 single DNA repair system.
There are several DNA repair systems that can fix different types of mutagen X.
If suspected mutagen X is affecting the rate, conduct a t-test.
The repair mecha nism is composed of a number of different proteins.
Two events are needed for DNA repair.
Identifying a mutagen is the topic.
The question is about analyzing the Ames test results.
An incorrect Ames test is recognized by a repair enzyme.
From your understanding of the topic, you can structure in the DNA and restore the remember that a higher number of colonies on the experimental correct structure.
Make a calculation.
The first thing you need to do to solve this problem is to use a template to make a normal strand of DNA.
There is a base pair mismatch in the DNA that can be repaired by applying the total number of cells to each not an abnormal nucleotide.
The mismatch is plate.
If the control and experimental data are removed, you need to conduct a t-test to recognize, and a strand of DNA in this region.
The strand is used to make a strand of DNA.
Statistics textbooks have a description of a t-test.
These are examples of other types of repair systems.
The abnormality is repaired in the second step.
The change in DNA structure can be repaired directly.
A new segment of DNA is created.
In this section, we will look at how the systems search for damaged DNA by examining the excision tracks.
This system is found in all species.
The undamaged strand is used as a template for the resynthesis of UvrAs when the damaged strand is removed.
NER can fix many different types of DNA damage, including UV-induced damage, chemically modified bases, missing bases, and various types of crosslinks.
It's best understood inbacteria.
In repairing damaged DNA, UvrC makes cuts on both sides.
The UvrC is out.
UvrD and UvrC are released and bind to UvrB at the site.
UvrB has been released.
The UvrC is able to make a hole in a strand of DNA.
UvrD has been released.
The UvrC is released after this process.
The strands of DNA are separated by UvrD.
The short DNA strand that contains the damaged region is removed by the action of UvrD.
UvrD has been released.
The gap is filled with the help of the polymerase and the ligase seals the undamaged strand as a template.
The original strand is made to by DNA ligase.
The NER system's components affect DNA repair.
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