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15 Microbial Mechanisms of Pathogenicity -- Part 6
The genitourinary tract is an important exit route.
We will look at a group of nonspecific defenses against disease in the next chapter.
It summarizes some key concepts anti- inflammatory drug, and they all recover from the mechanisms of the pathogenicity we have.
She holds a staff meeting to make sure the procedures for sterilizing are followed.
Adhesins are often associated with fimbriae.
It's important to study microbiology where it matters.
Mannose is the most common one.
Attachment and resistance to antimicrobial agents are provided by biofilms.
Pathogenicity is the ability of a pathogen to cause a disease.
Some pathogens have capsule that prevent them from being eaten.
The degree of pathogenicity is called virulence.
The specific route by which a pathogen gets access to another pathogen.
Many organisms can enter the body.
The disease can be spread by means of the kinases tracts.
Micro organisms cannot penetrate intact skin because they enter hair mucopolysaccharide and sweat ducts.
IgA antibodies are destroyed by IgA proteases.
The parenteral route is a route of penetration.
The host's immune system is affected by the expression of antigens by some microbes.
Many organisms can only cause infections when they have access to their specific portal of entry.
The actin of the host's cytoskeleton can be altered bybacteria.
Siderophores are used to get iron from the host.
Surface projections on a pathogen called adhesins adhere to the same receptors on the host.
Viruses grow inside the host's cells to avoid the host's immune response.
Viruses have attachment sites on the hostcel that allow them to gain access to it.
Toxemia is the presence of toxins in the blood.
There are visible signs of viral infections called cytopathic effects.
Some viruses cause cell death and others cause surrounding medium.
Damage but not death can be produced by exotoxins.
Antitoxins are produced against exotoxins.
A-B toxins have an active component and a binding component that attach the two parts to the target.
Cell lysis is caused by toxins that are disrupted.
Toxic shock syndrome toxin is one of the toxins that can cause the symptoms of fungal infections.
The host's genes are altered.
Endotoxins can be caused by host tissue or by the waste products of the parasites.
The host may be spared destruction by the host's antibodies if antibiotics, and cell death, are involved.
Humans are affected by endotoxins.
Pathogens leave a host.
There are genes for antibiotic resistance in plasmids.
The portal of exit for the microbes in blood is provided by the arthropods and syringes.
There are virulence factors that can result from lysogenic conversion.
The answers tab at the back of the textbook is where you can compare and contrast endotoxins.
Give an example of the toxins.
The diagram shows how the Shiga toxin enters and leaves the human body.
The capsule protozoa and helminths can cause an encapsulated bacterium to be deadly.
The 200 cells capsule does not contribute to virulence.
Skin is not the right portal of entry for smal pox.
The vaccine contained a mild form of the virus.
Smal pox can be transmitted by skin-to-skin contact.
Smal pox is a disease.
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